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Somatic mutations in chromatin remodeler genes could modify the epigenomic landscape of CLL, but They may be unusual With this malignancy in comparison with other lymphoid neoplasms. CHD2
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Environmental or self-antigens and homotypic interactions induce BCR and Toll-like receptor MBL77 (TLR) signaling, amplifying the response of CLL cells to other indicators through the microenvironment and growing the activation of anti-apoptotic and proliferation pathways.
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